Post Menopause women and Osteoporosis
Post Menopausal Osteoporosis in Women.
According to a Study of Women's Health Across the Nation (SWAN), we now have a far better grasp of how the female's bone health alters during menopausal transition (MT), expanding our awareness of a crucial period that has a significant impact on osteoporosis risk as people get older. One of the key sources for the review was the said study which provided extensive longitudinal assessments of bone health over MT.
After the age of 50, loss of muscle mass is significant along with similar gender-neutral changes like growing old, inflammation, decreased protein synthesis and myocyte regeneration, and several other gender-specific changes caused by the depletion of sex hormones. Muscle loss affects both sexes due to the decline in testosterone in males and estrogen in women. Numerous studies have shown a link between other age-related conditions and osteoporosis marked by fracture risk, the decreasing density of mineral and bone, bone tissue frailty, and muscle loss, and the presence of fragility factors or the bone mineral density (BMD) criteria is used to diagnose osteoporosis. Older people have more chances of developing osteoporosis, which affects women more frequently and often starts around menopause.
Osteoporosis (Arthritis, Rheumatoid Arthritis) is a condition which is characterized by the disruption of bone microarchitecture, bone tissue deterioration, and low bone mass: it can lead to compromised bone strength and an increased risk of fractures. It is a crippling ailment having noticeable adverse effects on your body, mind, the society, and the economy. The process of aging naturally brings the primary osteoporosis condition, and some systemic diseases and clinical pathologies cause secondary osteoporosis (Chronic bone pain and deformation of bones)
Estrogen insufficiency is linked to osteoporosis in postmenopausal age. This deficit causes growth in osteoclast activity and manufacture, which weakens the bone trabeculae and increases the fracture risk.
It is apparent that estrogen negatively affects osteoclast production and function since estrogen replacement therapy counteracts these effects, although it is unknown how estrogen achieves this..
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